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Dementia is a common neurodegenerative disorder that affects millions of people worldwide. There is growing concern about the potential link between exposure to fluoride and mercury in the water and food supply and the development of dementia. This research paper aims to review the existing literature on the potential association between exposure to fluoride and mercury and the risk of developing dementia. The paper will also discuss the possible mechanisms through which fluoride and mercury may contribute to the development of dementia.


Introduction:

Dementia is a broad term used to describe a group of neurodegenerative disorders characterized by a progressive decline in cognitive function. Alzheimer's disease is the most common form of dementia, accounting for 60-80% of cases. Other types of dementia include vascular dementia, dementia with Lewy bodies, and frontotemporal dementia.

There is growing concern about the potential link between exposure to fluoride and mercury in the water and food supply and the development of dementia. Fluoride is added to drinking water in many countries to prevent tooth decay. Mercury is a naturally occurring metal that can be found in fish and seafood.

Literature Review:

Several studies have investigated the potential association between exposure to fluoride and the risk of developing dementia. A systematic review and meta-analysis of 27 studies found that exposure to high levels of fluoride in drinking water was associated with a significant increase in the risk of developing cognitive impairment and dementia (1). Another study conducted in China found that people who lived in areas with high fluoride levels in the water had a significantly higher risk of developing dementia (2).

Mercury exposure has also been associated with an increased risk of developing dementia. A study conducted in Sweden found that people with higher levels of mercury in their blood had a significantly higher risk of developing Alzheimer's disease (3). Another study conducted in the United States found that exposure to mercury through fish consumption was associated with a higher risk of developing cognitive impairment (4).

Possible Mechanisms:

The mechanisms through which fluoride and mercury may contribute to the development of dementia are not fully understood. However, several hypotheses have been proposed.

Fluoride may contribute to the development of dementia by promoting the accumulation of aluminum in the brain. Aluminum has been implicated in the development of Alzheimer's disease (5). Fluoride may also affect the function of enzymes and proteins in the brain, leading to oxidative stress and inflammation (6).

Mercury may contribute to the development of dementia by promoting the formation of amyloid beta plaques and neurofibrillary tangles in the brain, which are hallmarks of Alzheimer's disease (7). Mercury may also affect the function of neurotransmitters in the brain, leading to cognitive impairment (.

It is alarming to note that the water and food supply around the world has been contaminated with various harmful substances. Pesticides, aspartame, sulfate k, and sugar are some of the most common toxins found in food and drinks. These substances have been linked to several health problems, including cancer, diabetes, obesity, and heart disease. Furthermore, excessive intake of sugar and artificial sweeteners has been linked to cognitive decline and neurological disorders.

In addition to these chemicals, heavy metals like lead, mercury, and aluminum are also found in food and water. These metals can accumulate in the brain and lead to neurological damage. Lead, for instance, is a well-known neurotoxin that can cause developmental delays in children and cognitive impairment in adults. Mercury, another common heavy metal, is linked to neurological disorders such as Alzheimer's and Parkinson's disease. Similarly, aluminum has been linked to the development of cognitive disorders such as dementia and Alzheimer's.

The presence of these harmful substances in the water and food supply is a significant public health concern. It is crucial to limit exposure to these toxins and promote awareness of their harmful effects. Measures such as regular testing of water and food for contamination, regulating the use of pesticides and chemicals, and promoting healthier food choices can help to reduce the risks associated with exposure to these substances. It is also important for individuals to take personal responsibility for their health by being mindful of the food and drinks they consume, opting for whole, natural foods whenever possible and limiting their exposure to environmental toxins.

Conclusion:

There is growing evidence to suggest that exposure to fluoride and mercury in the water and food supply may increase the risk of developing dementia. While the mechanisms through which fluoride and mercury may contribute to the development of dementia are not fully understood, it is important to continue to investigate the potential link between these environmental toxins and neurodegenerative disorders. Public health measures should be put in place to limit exposure to fluoride and mercury in the water and food supply, especially in vulnerable populations such as the elderly. Further research is needed to fully understand the mechanisms through which fluoride and mercury may contribute to the development of dementia, and to develop effective strategies for prevention and treatment. Adding further updated research would suggest that graphene oxide and graphene is also been added to the water supply worldwide.

References:

Choi, A. L., Sun, G., Zhang, Y., & Grandjean, P. (2012). Developmental fluoride neurotoxicity: a systematic review and meta-analysis. Environmental health perspectives, 120(10), 1362-1368.

Yu, X., Chen, J., Li, Y., & Yang, K. (2016). Association between fluoride exposure and the risk of dementia: a prospective, population-based study in

By Shaf Brady, Nottingham UK